The S1 sub-unit of the spike protein enters the parenchymal tissue of the brain in murine models. The brain’s endothelial cells attempt to hide the spike protein in the brain capillary glycocalyx, which can lead to degradation of the glycocalyx, dysfunction of the blood-brain barrier (BBB) and cerebral edema.
When these spike proteins cross the BBB, they induce neuron-inflammation and impair basic function of the central nervous system. A study from Nature Neuroscience finds the S1 spike protein of SARS-CoV-2 is engineered to attach to the angiotensin-converting enzyme-2 (ACE2), cross the BBB, and cause damage to the cardiovascular and central nervous systems. The spike protein can manipulate the central nervous system, causing an influx of cytokines, and setting off a cytokine storm. This influx of cytokines can also cross the blood-brain barrier, doing further damage to the immune response. When the viral binding protein crosses the blood-brain barrier, it enables the virus to cross the blood-brain barrier as well, allowing the virus to directly influence regions of the brain that regulate respiratory function. According to the study, injected spike proteins breach the BBB roughly ten times greater compared to intranasal exposure.
The spike protein primarily attaches to ACE2 but can attach to other proteins as well. The spike protein not only crosses the blood brain barrier, but it also travels through the bloodstream, causing blood clots. This toxin also attacks the olfactory bulb, liver, spleen and kidneys.....<<<<Read The Full Article Here>>>...
